Text book of veterinary medicine, Volume 2 (of 5) — Reading Companion

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Law, James, 1838-1921 Project Gutenberg 2023 Not confirmed
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Words 205,607
Reading time 894 min
Text sections 102

For Text book of veterinary medicine, Volume 2 (of 5) — Reading Companion, the stored edition analysis reports 205,607 words, 14 hr 54 min estimated reading time, and 102 detected text sections.

The text analysis averages about 19.8 words per sentence, while the detected sections provide another way to judge how the source is divided.

Project Gutenberg metadata also associates the work with “Veterinary medicine,” connecting these edition facts with the source record’s subject description.

This volume of James Law's veterinary textbook examines diseases of the digestive organs, liver, pancreas, and spleen in solipeds, ruminants, and omnivores, using comparative anatomy and detailed clinical observations from the early 1900s.
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ischarge of the fæcal matters, the air becomes more and more repulsive. The same odor pervades the mouth and the breath and the tongue is coated with a whitish, grayish or yellowish fur.

The fæces become more watery and slimy, with much casein in course of putrefaction, and the patient is rapidly run down by the profuse discharge and the general poisoning by absorbed putrid products. In the worst cases this may prove fatal in one or two days.

When the illness is more prolonged the alvine passages which at first number five or six per day, increase to fifteen or twenty and are passed with more effort, usually leaving the anus in a liquid stream. The color of the stools changes from a yellow to a grayish yellow or dirty white, hence the common name of _white scour_, and the fœtor is intensified.

Appetite may be in part preserved for a time but is gradually lost, and the subject becomes dull, listless and weak, indisposed to rise and walking unsteadily when raised. A general appearance of unthriftiness, staring coat, scurfy, unhealthy skin, pallor of the mucous membranes, arching of the back, tucking up and tenderness of the abdomen, excoriation of the margins of the anus, and congestion of the rectum as seen everted during defecation, mark the advance of the disease. Emaciation becomes very marked, and weakness and prostration extreme.

Fever usually sets in as the disease advances, as marked by hyperthermia, hot dry muzzle, hot ears, accelerated pulse and breathing.

When the intestinal fermentation is extreme there may be distinct bloating, more acute colicy pains, rumbling of the bowels and a frothy and even bloody condition of the dejections. The prostration may become extreme and the temperature reduced to the normal or below.

Death may result from inanition and exhaustion, or from nervous prostration and poisoning.

The affection may be complicated by purulent arthritis, peritonitis, pneumonia, hepatitis, keratitis or laminitis. It may prove fatal in from three to ten days.

_Mortality._ This is always high. For foals it has been set down at 80 per cent. of the numbers attacked, for calves at 54 to 90 per cent., and for lambs at 66 per cent. In 500 lambs Beresow found 300 attacked and 200 died. Kuleschow sets the losses at 30 to 40 per cent. of the lambs, and Cadeac advises that even the survivors should be fitted for the butcher as they are unfit for reproduction.

_Lesions._ In foals these are mainly confined to the intestines which show a more or less extended area of redness and congestion with catarrhal or pseudomembranous exudate on the mucosa, and the submucous connective tissue is infiltrated softened and marked by intense punctiform redness. The epithelium is swollen, opaque and easily detached, and Peyers patches infiltrated and prominent. The blood in the intestinal vessels is incoagulable. Exudation into the peritoneum and softening of the liver are not infrequent.

In calves the lesions are very similar, but the 4th stomach is usually implicated, the congestion and epithelial desquamation being most marked in the pyloric region. The contents of the intestines are mucopurulent, grayish, yellowish or red, and intensely fœtid. The follicles of the small intestine are red and projecting with an areola of congestion. Softening and even necrotic centers are found in the liver and kidneys and the mesenteric glands are swollen, red and softened.

In lambs the lesions are nearly the same in the 4th stomach, intestine, lymph glands, liver and kidneys. There is usually marked emaciation and the spleen and nerve centres are anæmic.

James Law opens Volume II by ranking digestive disorders second only to pulmonary complaints in solipeds and equally significant in ruminants. He immediately establishes a comparative framework, contrasting the gastro-intestinal surface area of carnivores (barely half the skin area) with herbivores (double to triple the skin area) to explain varying disease susceptibility. The text moves briskly from general principles to specific conditions, often beginning with a terse summary before expanding into causes, symptoms, lesions, and treatments. Law’s prose is clinical and direct, favoring concrete detail over rhetorical flourish.

Comparative Anatomy as Diagnostic Foundation

Law repeatedly anchors his discussion in anatomical differences. He notes that the carnivorous stomach is capacious relative to the intestine, allowing vomiting to expel irritants—a mechanism herbivores lack. For the horse, the gastro-intestinal surface is over double the skin area; in the ox, nearly triple. This structural variation, he argues, dictates both disease prevalence and clinical approach. When describing cryptogamic poisoning, he specifies that mouldy grains or beet pulp require large doses (about 150 to 200 lbs. a day) to prove deleterious, a figure that reflects his reliance on observed quantities rather than vague warnings. The comparative lens is not merely introductory but woven into each disease discussion, linking anatomy to prognosis.

Cryptogamic Poisoning: Moulds, Ergot, and Clinical Variation

Law devotes considerable space to poisoning from moulds, ergots, and smuts, cataloguing a range of sources: brewer’s grains, beet sugar marc, spoiled potatoes, mouldy bread, and musty fodder. He distinguishes between the slow development of symptoms from marc or ensilage—impaired appetite, salivation, tympany, colic, diarrhoea, debility, paresis, spasms, delirium—and the more dramatic gangrenous ergotism seen in Northern States winters. The description of ergotism is precise: necrotic sores with a hard, dry, black slough, and a pink or purple demarcation line. Law also notes that ergot toxicity varies with the stage of growth and water supply, showing an awareness of environmental factors unusual for the period. His treatment recommendations—potassium iodide, saline purgatives, oil of turpentine—reflect the era’s pharmacological toolkit.

Therapeutic Specifics and Cautionary Measures

Law’s therapeutic advice is practical and often detailed. For brain involvement in poisoning, he cites Cadeac’s recommendation of bleeding as both eliminating and sedative, then adds cold water or ice to the head, elevation, and doubled purgative doses. He stresses the importance of fixing the patient to a ring in the centre of a box stall to prevent self-injury—a concrete management detail. Prevention of cryptogamic poisoning involves salting and packing fresh grains or marc, a method he presents without fanfare. The text also includes warnings: mouldy bread can cause nervous disorders without digestive or urinary signs, a diagnostic pitfall. Law’s tone is that of a clinician sharing hard-won experience, not a theorist.

Structure of the Volume: From General to Specific

The volume’s organization moves from broad comparative physiology to individual diseases, each section following a consistent pattern: causes, symptoms, lesions, diagnosis, prevention, treatment. This structure allows rapid reference, though the density of information can be overwhelming. Law often begins a section with a bold-faced summary—e.g., “Moulds and bacteria in brewer’s grains… derange digestion, or cause abortion”—then expands. The text is punctuated by lists of symptoms and differential diagnoses (e.g., distinguishing cryptogamic poisoning from anthrax, coccidian hemorrhagic dysentery, foot and mouth disease, and rinderpest). These lists reflect the diagnostic challenges of the time, when laboratory confirmation was limited and clinical observation reigned.

Readers approaching this volume should expect a work rooted in early twentieth-century veterinary practice, where comparative anatomy and meticulous observation were the primary tools. Law’s emphasis on structural differences between species and his detailed cataloguing of poisoning sources offer a window into the era’s clinical challenges. The text rewards those who read systematically, as each section builds on the comparative framework established at the outset.

Old James Law’s volume, so full of careful aches in the gut, always puts me in mind of the quieter silences we keep for beings who cannot speak. That same attentive hand might reach toward Disease in captive wild mammals and birds — A Reader’s Guide, another patient ledger of suffering and hope, sitting close on my shelf, sharing bread and salt.

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